www.优德88.cpm ，基础医学与生物科学学院，生理学，教授，博导。2006年中国科学院动物研究所博士学位，主修方向，生殖生理及胚胎早期发育。2007-2012年博士后工作于英国伯明翰大学医学院，华威大学临床医学研究所及纽卡斯尔大学遗传医学研究中心。2012年入职www.优德88.cpm ，主要研究方向：干细胞/表观遗传，心肌发育毒性以及先心病的发病机制。主持课题：国家自然科学基金项目4项，苏州市科学基金项目1项。
中国生理学会教育工作委员会委员
中国动物学会细胞与分子显微技术分会，副秘书长，常务委员
江苏省发育生物学会理事
江苏省研究型医院学会生殖医学专委会，常务委员
江苏省生理学会理事
心肌发育毒性及先心病的发病机制；胚胎干细胞/表观遗传调控
生理学，解剖生理学
1.Huang Y#, Jiang B#, Xia Y, Wang J, Ji C, Tong J, Chen T*,Jiang Y*. Downregulation of miR-133a contributes to the cardiac developmental toxicity of trichloroethylene in zebrafish.Chemosphere.2020; 251:126610.
2.Jin H#, Ji C#, Ren F, Aniagu S, Tong J,Jiang Y*, Chen T*. AHR-mediated oxidative stress contributes to the cardiac developmental toxicity of trichloroethylene in zebrafish embryos.Journal of hazardous materials.2020; 385:121521.
3.Ren F, Wang J, Aniagu S, Li J,Jiang Y*, Chen T*. Effects of Trichloroethylene on the Expression of Long Intergenic Noncoding RNAs in B6C3F1 Mouse Liver.Chemical research in toxicology. 2020. Jan 27. [Epub ahead of print]
4.Jiang Y#, Li J#, Ren F, Ji C, Aniagu S, Chen T*. PM2.5-induced extensive DNA methylation changes in the heart of zebrafish embryos and the protective effect of folic acid.Environmental pollution. 2019;255(Pt 3):113331.
5.Chen T#, Jin H#, Wang H, Yao Y, Aniagu S, Tong J,Jiang Y*. Aryl hydrocarbon receptor mediates the cardiac developmental toxicity of EOM from PM2.5 in P19 embryonic carcinoma cells.Chemosphere. 2019; 216:372-8.
6.史佳林#,夏颖,花月,张可佳,陈涛*,姜岩*.三氯乙烯对斑马鱼胚胎心脏发育的毒性作用及机制.中国应用生理学杂志.2019;35(4):346-350.
7.Ren F#, Ji C#, Huang Y, Aniagu S,Jiang Y*, Chen T*. AHR-mediated ROS production contributes to the cardiac developmental toxicity of PM2.5 in zebrafish embryos.The Science of the total environment. 2019;719:135097.
8.金红梅#，任斐，乐聪，蔡怡萱，陈涛，姜岩*.三氯乙烯的心脏发育毒性.环境与职业医学.2018;35(1):14-8.
9.Yue C#, Ji C, Zhang H, Zhang LW, Tong J,Jiang Y*, Chen T*. Protective effects of folic acid on PM2.5-induced cardiac developmental toxicity in zebrafish embryos by targeting AhR and Wnt/beta-catenin signal pathways.Environmental toxicology. 2017;32(10):2316-22.
10.Jiang Y#*, Dai YF, Chen T*. Chapter Two - Advances in TCE Toxicology.Advances in Molecular Toxicology, Elsevier. 2017; 11:51-79.
11.Jiang Y#, Wang D#, Zhang G, Wang G, Tong J*, Chen T*. Disruption of cardiogenesis in human embryonic stem cells exposed to trichloroethylene.Environmental toxicology.2016;31(11):1372-
13.Jiang Y, Chen J, Yue C, Zhang H, Chen T*. Trichloroethylene-Induced DNA Methylation Changes in Male F344 Rat Liver.Chemical research in toxicology. 2016;29(10):1773-7.
14.Jiang Y#, Chen J#, Tong J, Chen T*. Trichloroethylene-induced gene expression and DNA methylation changes in B6C3F1 mouse liver.PloS one. 2014;9(12):e116179.
15.Jiang Y#, Habibollah S#, Tilgner K, Collin J, Barta T, Al-Aama JY, Tesarov L，Hussain R,
Trafford AW，Kirkwood G，Sernagor E，Eleftheriou CG，Przyborski S，Stojkovic M，Lako M，Keavney B，Armstrong L*. An induced pluripotent stem cell model of hypoplastic left heart syndrome (HLHS) reveals multiple expression and functional differences in HLHS-derived cardiac myocytes.Stem cells translational medicine. 2014;3(4):416-23.
16.Jiang Y#, Cowley SA#, Siler U#, Melguizo D, Tilgner K, Browne C, Dewilton, A，Przyborski, S，Saretzki, G，James WS，Seger RA，Reichenbach J，Lako M，Armstrong L*. Derivation and functional analysis of patient-specific induced pluripotent stem cells as an in vitro model of chronic granulomatous disease.Stem cells. 2012;30(4):599-611.
细胞生理（干细胞表观调控）
心血管生理（先心病致病机理）
努力是最美的风景，愿与你携手前行！
www.优德88.cpm ，基础医学与生物科学学院，生理学，教授，博导。2006年中国科学院动物研究所博士学位，主修方向，生殖生理及胚胎早期发育。2007-2012年博士后工作于英国伯明翰大学医学院，华威大学临床医学研究所及纽卡斯尔大学遗传医学研究中心。2012年入职www.优德88.cpm ，主要研究方向：干细胞/表观遗传，心肌发育毒性以及先心病的发病机制。主持课题：国家自然科学基金项目4项，苏州市科学基金项目1项。
中国生理学会教育工作委员会委员
中国动物学会细胞与分子显微技术分会，副秘书长，常务委员
江苏省发育生物学会理事
江苏省研究型医院学会生殖医学专委会，常务委员
江苏省生理学会理事
心肌发育毒性及先心病的发病机制；胚胎干细胞/表观遗传调控
生理学，解剖生理学
1.Huang Y#, Jiang B#, Xia Y, Wang J, Ji C, Tong J, Chen T*,Jiang Y*. Downregulation of miR-133a contributes to the cardiac developmental toxicity of trichloroethylene in zebrafish.Chemosphere.2020; 251:126610.
2.Jin H#, Ji C#, Ren F, Aniagu S, Tong J,Jiang Y*, Chen T*. AHR-mediated oxidative stress contributes to the cardiac developmental toxicity of trichloroethylene in zebrafish embryos.Journal of hazardous materials.2020; 385:121521.
3.Ren F, Wang J, Aniagu S, Li J,Jiang Y*, Chen T*. Effects of Trichloroethylene on the Expression of Long Intergenic Noncoding RNAs in B6C3F1 Mouse Liver.Chemical research in toxicology. 2020. Jan 27. [Epub ahead of print]
4.Jiang Y#, Li J#, Ren F, Ji C, Aniagu S, Chen T*. PM2.5-induced extensive DNA methylation changes in the heart of zebrafish embryos and the protective effect of folic acid.Environmental pollution. 2019;255(Pt 3):113331.
5.Chen T#, Jin H#, Wang H, Yao Y, Aniagu S, Tong J,Jiang Y*. Aryl hydrocarbon receptor mediates the cardiac developmental toxicity of EOM from PM2.5 in P19 embryonic carcinoma cells.Chemosphere. 2019; 216:372-8.
6.史佳林#,夏颖,花月,张可佳,陈涛*,姜岩*.三氯乙烯对斑马鱼胚胎心脏发育的毒性作用及机制.中国应用生理学杂志.2019;35(4):346-350.
7.Ren F#, Ji C#, Huang Y, Aniagu S,Jiang Y*, Chen T*. AHR-mediated ROS production contributes to the cardiac developmental toxicity of PM2.5 in zebrafish embryos.The Science of the total environment. 2019;719:135097.
8.金红梅#，任斐，乐聪，蔡怡萱，陈涛，姜岩*.三氯乙烯的心脏发育毒性.环境与职业医学.2018;35(1):14-8.
9.Yue C#, Ji C, Zhang H, Zhang LW, Tong J,Jiang Y*, Chen T*. Protective effects of folic acid on PM2.5-induced cardiac developmental toxicity in zebrafish embryos by targeting AhR and Wnt/beta-catenin signal pathways.Environmental toxicology. 2017;32(10):2316-22.
10.Jiang Y#*, Dai YF, Chen T*. Chapter Two - Advances in TCE Toxicology.Advances in Molecular Toxicology, Elsevier. 2017; 11:51-79.
11.Jiang Y#, Wang D#, Zhang G, Wang G, Tong J*, Chen T*. Disruption of cardiogenesis in human embryonic stem cells exposed to trichloroethylene.Environmental toxicology.2016;31(11):1372-
13.Jiang Y, Chen J, Yue C, Zhang H, Chen T*. Trichloroethylene-Induced DNA Methylation Changes in Male F344 Rat Liver.Chemical research in toxicology. 2016;29(10):1773-7.
14.Jiang Y#, Chen J#, Tong J, Chen T*. Trichloroethylene-induced gene expression and DNA methylation changes in B6C3F1 mouse liver.PloS one. 2014;9(12):e116179.
15.Jiang Y#, Habibollah S#, Tilgner K, Collin J, Barta T, Al-Aama JY, Tesarov L，Hussain R,
Trafford AW，Kirkwood G，Sernagor E，Eleftheriou CG，Przyborski S，Stojkovic M，Lako M，Keavney B，Armstrong L*. An induced pluripotent stem cell model of hypoplastic left heart syndrome (HLHS) reveals multiple expression and functional differences in HLHS-derived cardiac myocytes.Stem cells translational medicine. 2014;3(4):416-23.
16.Jiang Y#, Cowley SA#, Siler U#, Melguizo D, Tilgner K, Browne C, Dewilton, A，Przyborski, S，Saretzki, G，James WS，Seger RA，Reichenbach J，Lako M，Armstrong L*. Derivation and functional analysis of patient-specific induced pluripotent stem cells as an in vitro model of chronic granulomatous disease.Stem cells. 2012;30(4):599-611.
细胞生理（干细胞表观调控）
心血管生理（先心病致病机理）
努力是最美的风景，愿与你携手前行！